Another cause is Dressler syndrome which occurs several weeks after a myocardial infarction, or heart attack.

另一个病因是发生于肌梗死数周后的肌梗死后综合征。

And that can lead to a myocardial infarction, which is what we also call a heart attack.

这可能导致肌梗塞，也就是我们所说的脏病发作。

An MI means myocardial infarction, just another name for a heart attack.

MI 表示肌梗塞，只是脏病发作的别。

Okay, it's not an infarction. It's a dissection.

好吧 这不是肌梗塞 是夹层。

Basically, when heart cells die in a myocardial infarction, it leads to massive inflammation that also involves the serous pericardium.

肌梗死导致肌细胞死亡，引发累及膜包的强烈炎症反应。

In contrast partial occlusion may progress to localized infarction.

相反，部分闭塞可发展为局部梗死。

The susceptibility of the ventricular wall to this type of partial thickness infarction is not uniform.

室壁对这种部分厚度梗死的易感性并不一致。

It should be noted, however, that this condition is clinically unstable and often precedes a myocardial infarction.

肌肌钙蛋白水平保持正常，或只是轻微升高。然而，应该指出的是，这种疾病在临床上是不稳定的，而且常常发生在肌梗死之前。

In severe cases, blood flow can been completely cut off, which results in myocardial infarction, or the necrosis of a portion of myocardium.

严重的情况下，血流会被完全阻断，引起肌梗死，即肌的部分坏死。

Okay, let's recap - acute pericarditis is usually caused by a idiopathic causes, but can be caused by viruses or after a myocardial infarction.

急性包炎通常原因未明，也可因病毒感染或肌梗死导致。

Then entire thickness of the myocardial wall supplied by that vessel become ischemic, and is at risk of infarction.

然后，由该血管供应的整个肌壁就会缺血，具有梗死的风险。

In the absence of treatment, the process may progress to complete transmural infarction of the area involved with development of pathological Q waves.

在没有治疗的情况下，这一过程可能进展到与病理性Q波发展有关的区域的完全跨壁梗死。

A prolonged ischemia can lead to death of the heart tissue, known as heart attack or myocardial infarction.

血液供应减少为缺血。长时间缺血可导致脏组织死亡，即脏病发作或肌梗死。

The ECG changes associated with infarction in this situation are identical to those of pure ischemia with depression of the ST segment, T-wave inversion or both.

这种情况下与梗死相关的电图变化与单纯缺血伴ST段压低、T波倒置或两者兼而有之的电图变化相同。

So we've seen that in ST elevation MI, untreated a complete arterial occlusion may progress to infarction of the full thickness of the affected myocardial wall.

所以我们已经看到，在ST段抬高型肌梗死中，未经治疗的完全性动脉闭塞可能发展为肌壁全厚度梗死。

STEMI is infarction that extends the entire thickness of the myocardium.

STEMI指肌梗死延伸至肌全层。

In inferior infarction, the injury current represented here by the blue arrows, therefore produces ST elevation in these leads.

下壁梗死时，受损电流用蓝色箭头表示，因此在这些导联产生ST段抬高。

The location of infarction can be identified based on the leads with ST elevation.

通常在电相反导联上有一个相互的ST段压低。

As shown here, as the process of infarction evolves the T waves may demonstrate a further abnormality developing ash a variable time point after occlusion.

如图所示，随着梗死过程的发展，T波可能在堵塞后任一时间点出现进一步的异常发展。

Most patients with infarction show elevated troponin levels within 6 hours.

大多数脑梗死患者在6小时内肌钙蛋白水平升高。