Now if this inactivation's random, you'd expect about half of the female's cells to have a functional dystrophin gene and the other half to have a defective dystrophin gene, and these people are typically asymptomatic.

如果失活是随机的，可预料到女性半的细胞中抗肌萎缩蛋白因是正常的，另半是有缺陷的，通常这些人无临床症状。

Women with one mutated gene copy have a remaining healthy copy, so they don't get hemophilia unless X-chromosome inactivation turns off the normal copy in the majority of cells.

携带突变因的女性还有健康的因拷贝，所们般不会患血友病，除非X染色体失活使得大多数细胞中正常的因拷贝沉默。

Currently, how people usually look at it, we have to give more surfactant, we have to revert this inactivation, right?

目前， 人们普遍认为，我们得给予更多的表面活性物质， 必须逆转这种失活，对吧？

The embryo split into twins before X inactivation, so each twin ended up with a different inactivation pattern.

胚胎在X染色体失活前分裂成双胞胎，因此每双胞胎最终形成了不同的失活模式。

When these early cells divide, each passes on its X inactivation.

当这些早期细胞分裂时，每细胞都会传递其X染色体失活状态。

There are still unresolved questions about X inactivation, like how some genes on the X chromosome escape inactivation and why inactivation isn't always random.

关于X染色体失活， 仍存在些未解之谜，例如，某些位于X染色体上的因如何逃脱失活机制， 及为何失活过程并非总是随机发生。