1.The result is a reduced excitability or responsiveness of the neurons and muscles in the heart.

这导致心脏神经元和肌肉的兴奋或反应降低。

2.The effect this has on excitability of myocytes, however, is complex.

然而，这对心肌细胞兴奋的影响是复杂的。

3.While initial changes seem to increase myocyte excitability; further rise of potassium has the OPPOSITE effect.

化可能增加心肌细胞的兴奋，但钾进一步升高却具有相反的作用。

4.This reduces the heart's excitability and suppresses reentrant tachycardia.

这降低心脏的兴奋和抑制折返快速心律失常。

5.Now, as you may know, the binding of glutamate to the NMDA receptor results in an influx of extracellular calcium, which controls membrane excitability and synaptic transmission.

现在，如你所知，谷氨酸与NMDA受体的结合导致内流细胞外钙，控制膜的兴奋和突触传递。