acidosis

Hematologic studies revealed severe hyperphosphatemia, hypocalcemia and metabolic acidosis.

血液显示有严重高磷血症、低钙血症和代谢性中毒。

Amino acids and other organic acids – contribute to acidosis, aciduria, and dysrhythmia.

氨基和其他有机—中毒，尿和节律障碍。

Hypercalciuria was considered as a secondary condition when (It was) associated with familial renal tubular acidosis.

高钙尿如伴有家族性小管性中毒，就可以认为是继发病。